HEL — Solanum tuberosum
Summary
PGSC0003DMG400019435, designated HEL and also known as WIN1, encodes wound-induced protein WIN2, a member of the PR4 (pathogenesis-related protein 4) family that contains a Barwin domain and a chitin-binding domain. The gene product exhibits RNA nuclease activity, chitin binding, sugar binding, hydrolase activity, transmembrane receptor protein serine/threonine kinase activity, and protein binding. HEL is associated with responses to abiotic stimulus, herbivore, virus, and stress, as well as systemic acquired resistance, regulation of hormone levels, leaf abscission, vascular transport, phloem transport, leaf vascular tissue pattern formation, and killing of cells of other organisms. The protein localizes to secretory vesicles, the extracellular region, and the plasma membrane. The gene is associated with leaf vein morphology, root length, plant height, pathogen defense response, wilt symptom development, resistance to Botrytis cinerea, salt tolerance, premature senescence, leaf cell death, and abnormal leaf venation.
Annotations
- Chromosome
- 1
- Related genes
- HEL, VH1
- GO terms
- Vascular Transport, NO storage, killing of cells of other organism, Secretory Vesicle, Response To Ethylene, Abscission, Systemic Acquired Resistance, HR-PCD
- Publications
- 12668776, 25643917, 25261123, 22731664, 23234406, 23448426, 16081527, PMID:19857612
- Traits
- antifungal activity, Premature Senescence, Leaf Cell Death, Abnormal Leaf Venation, Reduced Vein Number, vascular development, pathogen defense response, seed germination under salt stress, salt tolerance, insect resistance