HEL — Solanum tuberosum

Summary

PGSC0003DMG400019435, designated HEL and also known as WIN1, encodes wound-induced protein WIN2, a member of the PR4 (pathogenesis-related protein 4) family that contains a Barwin domain and a chitin-binding domain. The gene product exhibits RNA nuclease activity, chitin binding, sugar binding, hydrolase activity, transmembrane receptor protein serine/threonine kinase activity, and protein binding. HEL is associated with responses to abiotic stimulus, herbivore, virus, and stress, as well as systemic acquired resistance, regulation of hormone levels, leaf abscission, vascular transport, phloem transport, leaf vascular tissue pattern formation, and killing of cells of other organisms. The protein localizes to secretory vesicles, the extracellular region, and the plasma membrane. The gene is associated with leaf vein morphology, root length, plant height, pathogen defense response, wilt symptom development, resistance to Botrytis cinerea, salt tolerance, premature senescence, leaf cell death, and abnormal leaf venation.

Annotations

Chromosome
1
Related genes
HEL, VH1
GO terms
Vascular Transport, NO storage, killing of cells of other organism, Secretory Vesicle, Response To Ethylene, Abscission, Systemic Acquired Resistance, HR-PCD
Publications
12668776, 25643917, 25261123, 22731664, 23234406, 23448426, 16081527, PMID:19857612
Traits
antifungal activity, Premature Senescence, Leaf Cell Death, Abnormal Leaf Venation, Reduced Vein Number, vascular development, pathogen defense response, seed germination under salt stress, salt tolerance, insect resistance