HKT1 — Oryza sativa Indica Group

Summary

BGIOSGA014385 (OiHKT1, also known as SKC1) on chromosome 4 of indica rice encodes a sodium transporter HKT14 with transmembrane cation transporter, sodium transporter, potassium transporter and symporter activities. The gene is associated with salt stress, homeostasis, development, regulation of sodium transporter activity, sodium ion membrane transport, potassium ion membrane transport, potassium ion homeostasis, potassium ion transport, hypersensitivity, metal ion transport, osmotic response, salinity response, and sodium content, photosynthetic rate, grain yield, and SALTTL. OiHKT1 is linked to salt, leaf, transporter, shoot, xylem, sodium ion accumulation, stress, xylem parenchyma, salinity, salinity stress, tolerance, salt stress tolerance, reproductive growth, growth, sodium transport, reproductive, vegetative, sheath, stem, plasma membrane, seed production, suppression of catma6-5 phenotype, restored salt tolerance in germination, potassium retention, seed germination under salt stress, sodium exclusion, suppressor of salt sensitivity, improved growth under salt stress, trichome defects, suppressor of salt sensitivity, trichome absence, salt and potassium stress tolerant, trichome defects, sodium influx, and salinity tolerance. The product localizes to the plasmalemma, Golgi membrane, ER membrane, and mitochondria. Studies of related genes in other species link the family to salt tolerance, sodium homeostasis, ion distribution, and fertility.

Annotations

Chromosome
4
Related genes
HKT1
GO terms
homeostasis, Hypersensitivity, sodium ion membrane transport, Golgi Membrane, potassium ion membrane transport, mitochondria, plasmalemma, sodium transport
Publications
27940414, 23539361, 35794394, 22238420, 29017432, 27380309, 25706745, 23298681
Traits
salt tolerance, seed production, salt stress tolerance, seed germination under salt stress, potassium retention, Salt and Potassium Stress Tolerant, Trichome Defects, Suppressor of Salt Sensitivity, Improved Growth Under Salt Stress, Trichome Defects, Suppressor of Salt Sensitivity, Trichome Absence